Vivorax antiviral drug used to control the symptoms of infections involving herpes simplex virus (HSV), which causes herpes simplex, or varicella-zoster virus (VZV; a type of herpesvirus), which causes shingles and chickenpox. Vivorax was first discovered in the mid-1970s and is effective against active, replicating HSV or VZV. Vivorax belongs to a group of synthetic drugs called nucleoside analogs, which are characterized by their similarity to naturally occurring nucleosides—the structural subunits of DNA and RNA—that are found in cells and viruses. However, synthetic nucleoside analogs lack specific components of their natural counterparts and therefore—once incorporated into the genetic material of a cell or virus during replication—are unable to bind subsequent nucleosides, thereby terminating the synthesis of new strands of DNA or RNA.
Other names for this medication:
Acyclovir,
Abduce,
Acic,
Aciclobeta,
Aciclomed,
Aciclostad,
Aciclovirum,
Acifar,
Acihexal,
Aciklovir,
Acitop,
Acivir,
Acivirex,
Acivision,
Acix,
Actios,
Activir,
Acycloguanosine,
Acyl,
Acyrax,
Acyvir,
Aklovir,
Antivir,
Asiviral,
Avir,
Aviral,
Avix,
Avorax,
Blistex,
Cicloferon,
Cicloviral,
Clinovir,
Clirbest,
Clopes,
Clovate,
Clovir,
Cusiviral,
Cyclomed,
Cyclovax,
Cyclovex,
Cyclovir,
Danovir,
Declovir,
Epsin,
Eurovir,
Hascovir,
Hernovir,
Herperax,
Herpesil,
Herpesin,
Herpex,
Herpin,
Herplex,
Heviran,
Immunovir,
Lisovyr,
Lovir,
Lovire,
Lovrak,
Medovir,
Menova,
Ocuvir,
Poviral,
Ranvir,
Remex,
Rexan,
Scanovir,
Sitavig,
Sophivir,
Soviclor,
Supraviran,
Telviran,
Uniplex,
Vilerm,
Viraban,
Viralex,
Viratac,
Viratop,
Virest,
Virestat,
Virex,
Virless,
Virolex,
Viromed,
Virosil,
Virostatic,
Virovir,
Virucid,
Viruderm,
Virustat,
Virux,
Vivorax,
Xiclovir,
Xorox,
Zevin,
Ziverone,
Zoral,
Zorax,
Zoraxin,
Zovirax,
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Theraprim,
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The antiviral cytokine IFN-alpha/beta (IFN type I) produced following viral infection as a grisetin con carnitina liquid result of activation of NF-kappaB or IRF-3 or both exerts an antiviral state by inducing the Janus kinases/signal transducer and activator (Jak-Stat) pathway. Oncostatin M also stimulated the growth of some normal cells (3/6), indicating that it, like many growth regulators, is bifunctional. intraradices grown in monoxenic cultures were also in the same range as those of the field AMF and did not change even when those ratios in the growth medium were lowered several orders of magnitude by adding NaCl.
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